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“The thermooxidative aging of ammonia-catalyzed phenolic resin for 30 days at 60170 degrees C was investigated in this article. The aging mechanism and thermal properties of the phenolic resin during thermooxidative aging were described by thermogravimetry (TG)Fourier transform infrared (FTIR) spectroscopy, attenuated total reflectance (ATR)FTIR spectroscopy, and dynamic mechanical thermal analysis. The results
show that the C?N bond decomposed into ammonia and the dehydration condensation between the residual hydroxyl groups occurred during the thermooxidative aging. Because of the presence Belnacasan of oxygen, the m(e)thylene bridges were oxidized into carbonyl groups. After aging for 30 days, the mass loss ratio reached 4.50%. The results of weight change at high temperatures coincided with the results of TGFTIR spectroscopy and ATRFTIR spectroscopy. The glass-transition temperature (Tg) increased from 240 to 312 degrees C after thermooxidative aging for 30 days, which revealed the postcuring of phenolic resins. In addition, an empirical equation between the weight change ratio and Tg was obtained. (c) 2012 Wiley Periodicals, Inc. J Appl Polym Sci, 2012″
“The authors begin with a discussion of the anatomy relevant to palatoplasty. Perioperative considerations are then addressed. A broad range of surgical options has evolved over
time; these are discussed in their historical context. The authors present a detailed description of their preferred surgical approach. Postoperative care
is then described. An examination of recent trends and controversies in the click here field is then FRAX597 inhibitor offered. Finally, an approach to outcomes assessment is discussed. It is hoped that this monograph will be of use in guiding others as they embark on the highly challenging, but equally rewarding, task of perfecting the palatoplasty.”
“Renin-Angiotensin System (RAS) plays an important role in the development of Metabolic Syndrome (MS) and in aging. Angiotensin 1-7 (Ang 1-7) has opposite effects to Ang II. All of the components of RAS are expressed locally in adipose tissue and there is over-activation of adipose RAS in obesity and hypertension. We determined serum and abdominal adipose tissue Ang II and Ang 1-7 in control and MS rats during aging and the expression of AT1, AT2 and Mas in white adipose tissue. MS was induced by sucrose ingestion during 6, 12 and 18 months. During aging, an increase in body weight, abdominal fat and dyslipidemia were found but increases in aging MS rats were higher. Control and MS concentrations of serum Ang II from 6-month old rats were similar. Aging did not modify Ang II seric concentration in control rats but decreased it in MS rats. Ang II levels increased in WAT from both groups of rats. Serum and adipose tissue Ang 1-7 increased during aging in MS rats.